Kcnq1 (Kvlqt1, a potassium channel involved in human long QT syndrome) is preferentially maternally
expressed in fetal mice (except brain) but biallelically expressed from the neonatal and late
juvenile stages (Gould
TD and Pfeifer K 1998; Paulsen
M et al, 1998).
Mancini-Dinardo et al. showed lack of a full-length Kcnq1ot1 transcript on
the paternal chromosome leads to the expression of genes that are normally paternally repressed (Mancini-Dinardo
et al, 2006 ) .